What does depressed st segment mean
Basic Concept: the specificity of ST-T and U wave abnormalities is provided more by the clinical circumstances in which the ECG changes are found than by the particular changes themselves. Thus the term, nonspecific ST-T wave abnormalities , is frequently used when the clinical data are not available to correlate with the ECG findings.
This does not mean that the ECG changes are unimportant! We also use third-party cookies that help us analyze and understand how you use this website. These cookies will be stored in your browser only with your consent. You also have the option to opt-out of these cookies. But opting out of some of these cookies may have an effect on your browsing experience. Necessary Necessary. Necessary cookies are absolutely essential for the website to function properly.
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Advertisement advertisement. Advertisement cookies are used to provide visitors with relevant ads and marketing campaigns. These cookies track visitors across websites and collect information to provide customized ads. Current guideline criteria for ischemic ST segment depression:. The transition from ST segment to T-wave is more abrupt in ischemia the transition is normally smooth.
Refer to Figure 2. ST segment depressions with upsloping ST segment are rarely caused by ischemia, with one notable exception. Occurrence of upsloping ST segment depressions with prominent T-waves in the majority of the chest leads may indicate an acute occlusion in the LAD left anterior descending artery.
This figure must be studied carefully. Normal physiological ST segment depressions occur during physical exercise. These ST segment depressions have an upsloping ST segment.
Some experts believe that these ST segment depressions represent a benign form of subendocardial ischemia. Refer to Exercise Stress Testing for details.
Hyperventilation cause ST segment depressions very similar to those normally seen during physical exercise. These are all common conditions in which an abnormal depolarization QRS complex causes abnormalities in the repolarization ST-T-segment. For example, a block in the left bundle branch i. The abnormal ventricular depolarization will cause an abnormal repolarization.
It is actually expected that these conditions display such secondary ST-T changes; absence of such changes should lead to suspicion of ischemia if the patient has symptoms consistent with ischemia. The same is true for artificial pacemakers virtually all pacemakers stimulate the ventricles in the right ventricular apex.
Thus, it is expected to observe secondary ST-T changes during pacemaker rhythm. Sympathetic stimulation and hypokalemia causes non specific ST segment changes.
These depressions are horizontal or downsloping.
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